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公共卫生学院
硕士生导师简介-朱利娟
2025-03-05 14:14  

南华大学公共卫生学院研究生导师简介


名:

朱利娟

出生年月

19914

位:

博士研究生

职称/职务:

讲师

籍贯:

湖南省娄底

研究方向:

营养与健康


一、基本情况

南华大学公共卫生学院营养与食品卫生学教研室教师,博士,讲师,硕士生导师。主要从事营养代谢相关的研究。主持湖南省教育厅一般项目1项,参加湖南省重点研发计划、湖南省自然科学基金、湖南省发展与改革委员会创新平台建设项目等多项研究课题。目前已发表学术论文共计30余篇,其中以第一作者或通讯作者在《Food & Function》、《Ecotoxicology and Environmental Safety》、《Journal of Functional Foods》、《Antioxidants》、《Biomedicine & Pharmacotherapy》、《Toxins》、《International Immunopharmacology》、《Journal of Food Biochemistry》等杂志上发表SCI论文10篇,以第一作者或通讯作者发表中文核心期刊6


(一)教育背景

2009.9-2013.6 湖南农业大学,动物医学,学士

2014.9-2017.6 湖南农业大学,临床兽医学,硕士,导师:易金娥教授

2019.9 -2023.6 湖南农业大学兽医学,博士,导师:易金娥教授


(二)工作经历

2017.7-2019.8 长沙绿叶生物科技有限公司研发专员

       2023.7-今 南华大学公共卫生学院营养与食品卫生学教研室,预防医学教学与科研工作,讲师


二、教学与科研情况

)科研项目

1

湖南省教育厅,一般项目,2324JY189靶向铁死亡探究苏木酮A缓解炎症性肠病的作用及分子机制2024-012026-121万元,主持。

2

湖南省科技厅,湖南省重点研发计划,2020NK2032,畜禽主要非传染性群发病防控关键技术研究,2021-012022-1250万元,参与。

3

湖南省科技厅,湖南省自然科学基金面上项目,2020JJ4368,基于ROS-NLRP3炎性通路研究桦木酸缓解断奶仔猪肠道炎症的作用机制,2020-012022-125万元,参与。

4

湖南省发展与改革委员会,创新研发项目,202114,基于Nrf2/MAPK信号通路研究桦木酸缓解玉米赤霉烯酮致睾丸氧化损伤的保护机制,2021-032022-1210万元,参与。

5

湖南省发展与改革委员会,创新研发项目,201937,基于NF-κB/MAPK/ Nrf2-HO-1信号通路研究桦木酸缓解T-2毒素致肠黏膜氧化损伤的保护机制,2019-092021-0810万元,参与。


)代表性科研和教改论文

1.代表性科研论文

1

He E#, Ma Y#, Kong L, Huang Y, Huang C, Yang W, Yi J*, Zhu L*. Suppression of endoplasmic reticulum stress-associated pathways and hepatocyte apoptosis participates in the attenuation of betulinic acid on alcohol-provoked liver injury in mice. Food & Function, 2022, 13(22): 11489-11502.通讯作者

2

Ma Y#, Xiang S#, Jiang W, Kong L, Tan Z, Liang Z, Yuan Z, Yi J*, Zhu L*. Gamma-oryzanol protects human liver cell (L02) from hydrogen peroxide-induced oxidative damage through regulation of the MAPK/Nrf2 signaling pathways. Journal of Food Biochemistry, 2022: e14118.通讯作者

3

Zhu L#, Luo C#, Ma C, Kong L, Huang Y, Yang W, Huang C, Jiang W*, Yi J*. Inhibition of the NF-κB pathway and ERK-mediated mitochondrial apoptotic pathway takes part in the mitigative effect of betulinic acid on inflammation and oxidative stress in cyclophosphamide-triggered renal damage of mice. Ecotoxicology and Environmental Safety, 2022, 246: 114150.

4

Zhu L#, Yi X#, Ma C, Luo C, Kong L, Lin X, Gao Y, Yuan Z, Wen L, Li R, Wu J*, Yi J*. Betulinic acid attenuates oxidative stress in the thymus induced by acute exposure to T-2 toxin via regulation of the MAPK/Nrf2 signaling pathway. Toxins, 2020, 12(9): 540.

5

Zhu L#, Xiang S#, Ou Z, Ma C, Yi X, Huang L, Ma Y, Liang Z, Yuan Z, Wen L, Li R, Wu J, Tan Z*, Yi J*. Gamma-oryzanol prevents ethanol-induced liver injury by ameliorating oxidative stress and modulating apoptosis-related protein expression in mice. Journal of Functional Foods, 2019, 62, 103532.

6

Zhu L, Yi X, Zhao J, Yuan Z, Wen L, Pozniak B, Obminska-Mrukowicz B, Tian Y, Tan Z, Wu J*, Yi J*. Betulinic acid attenuates dexamethasone-induced oxidative damage through the JNK-P38 MAPK signaling pathway in mice. Biomedicine & Pharmacotherapy, 2018, 103: 499-508.

7

Lin X#, Zhu L#, Gao X, Kong L, Huang Y, Zhao H, Chen Y, Wen L, Li R, Wu J, Yuan Z*, Yi J*. Ameliorative effect of betulinic acid against zearalenone exposure triggers testicular dysfunction and oxidative stress in mice via p38/ERK MAPK inhibition and Nrf2-mediated antioxidant defense activation. Ecotoxicology and Environmental Safety, 2022, 238: 113561.共同一作

8

Kong L#, Zhu L#, Yi X, Huang Y, Zhao H, Chen Y, Yuan Z, Wen L, Wu J*, Yi J*. Betulinic acid alleviates spleen oxidative damage induced by acute intraperitoneal exposure to T-2 toxin by activating Nrf2 and inhibiting MAPK signaling pathways. Antioxidants, 2021, 10(2): 158.共同一作

9

Ou Z#, Zhu L#, Huang C, Ma C, Kong L, Lin X, Gao X, Huang L, Wen L, Liang Z, Yuan Z, Wu J*, Yi J*. Betulinic acid attenuates cyclophosphamide-induced intestinal mucosa injury by inhibiting the NF-κB/MAPK signalling pathways and activating the Nrf2 signalling pathway. Ecotoxicology and Environmental Safety, 2021, 225: 112746.共同一作

10

Huang L#, Zhu L#, Ou Z, Ma C, Kong L, Huang Y, Chen Y, Zhao H, Wen L, Wu J, Yuan Z*, Yi J*. Betulinic acid protects against renal damage by attenuation of oxidative stress and inflammation via Nrf2 signaling pathway in T-2 toxin-induced mice. International Immunopharmacology, 2021, 101: 108210.共同一作

11

Kong L#, Gao X#, Zhu L, Lin X, Huang Y, Huang C, Yang W, Chen Y, Zhao H, Wu J, Yuan Z*, Yi J*. Betulinic acid protects against ovarian impairment by decreasing F-2 toxin-induced oxidative stress and inflammation associated with the downregulation of p38 expression in mice. Food Science and Human Wellness, 2024, 13(3): 1292-1302.

12


Ou Z#, Zhao J#, Zhu L, Huang L, Ma Y, Ma C, Luo C, Zhu Z, Yuan Z, Wu J, Li R*, Yi J*. Anti-inflammatory effect and potential mechanism of betulinic acid on λ-carrageenan-induced paw edema in mice. Biomedicine & Pharmacotherapy, 2019, 118: 109347.

13

Huang L#, Jiang W#, Zhu L, Ma C, Ou Z, Luo C, Wu J, Wen L, Tan Z*, Yi J*. γ-Oryzanol suppresses cell apoptosis by inhibiting reactive oxygen species-mediated mitochondrial signaling pathway in H2O2-stimulated L02 cells. Biomedicine & Pharmacotherapy, 2020, 121: 109554.

14

Luo C#, Huang C#, Zhu L, Kong L, Yuan Z, Wen L, Li R, Wu J*, Yi J*. Betulinic acid ameliorates the T-2 toxin-triggered intestinal impairment in mice by inhibiting inflammation and mucosal barrier dysfunction through the NF-κB signaling pathway. Toxins. 2020, 12(12): 794.

15

Wang X#, Yuan Z#, Zhu L, Yi X, Ou Z, Li R, Tan Z, Pozniak B, Obminska-Mrukowicz B, Wu J*, Yi J*. Protective effects of betulinic acid on intestinal mucosal injury induced by cyclophosphamide in mice. Pharmacological Reports, 2019, 71(5): 929-939.

、联系方式

电话:18874205749

        E-mailzhulijuan@usc.edu.cn


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